severe chest pain, rest and nitrates can not be completely alleviated, with increased white blood cells, fever, accelerated erythrocyte sedimentation rate, serum enzyme activity increased and the progressive ECG changes, may be complicated by arrhythmia, shock, or effort failure and other complications, often life-threatening. The disease most common in Europe and America, about 150 million people each year in the United States myocardial infarction. China in the world is a low incidence area, but also on the rise in recent years.
Myocardial infarction, coronary artery occlusion blood flow interruption, so that part of the persistence of myocardial ischemia due to severe local necrosis occurs. Clinically more severe and persistent chest pain, fever, leukocytosis, erythrocyte sedimentation rate, which increased serum enzyme activity and the progressive ECG changes, arrhythmias may occur, shock or heart failure.
Coronary atherosclerosis on the basis of plaque rupture complicated endovascular thrombosis or arterial intimal hemorrhage sustained spasm lasting rapidly and completely the lumen of the occlusion, such as the coronary arteries and other inter- previously not fully established collateral circulation in the arteries that supply can lead to severe myocardial ischemia lasting more than 1 hour that is induced myocardial necrosis in coronary atherosclerotic stenosis based on the occurrence of sudden drop in cardiac output (hemorrhagic shock or serious arrhythmias), or left ventricular load surge (severe physical activity, a sudden rise in blood pressure and emotional over-excitement or straining at stool), also can cause myocardial ischemia and myocardial necrosis in severe persistent meal (especially when eating large amounts of fat) increased blood lipid increased blood viscosity caused by local blood flow is slow platelet aggregation Erzhi easy thrombosis; sleep increased vagal tone during coronary artery spasm can be increased to an extent which rendered necrotic myocardial ischemia can occur in patients with frequent angina may also be in the original and in asymptomatic
If it were not itself have had congenital heart disease before myocardial ischemia should be!
Myocardial infarction, coronary artery occlusion blood flow interruption, so that part of the persistence of myocardial ischemia due to severe local necrosis occurs. Clinically more severe and persistent chest pain, fever, leukocytosis, erythrocyte sedimentation rate, which increased serum enzyme activity and the progressive ECG changes, arrhythmias may occur, shock or heart failure.
Coronary atherosclerosis on the basis of plaque rupture complicated endovascular thrombosis or arterial intimal hemorrhage sustained spasm lasting rapidly and completely the lumen of the occlusion, such as the coronary arteries and other inter- previously not fully established collateral circulation in the arteries that supply can lead to severe myocardial ischemia lasting more than 1 hour that is induced myocardial necrosis in coronary atherosclerotic stenosis based on the occurrence of sudden drop in cardiac output (hemorrhagic shock or serious arrhythmias), or left ventricular load surge (severe physical activity, a sudden rise in blood pressure and emotional over-excitement or straining at stool), also can cause myocardial ischemia and myocardial necrosis in severe persistent meal (especially when eating large amounts of fat) increased blood lipid increased blood viscosity caused by local blood flow is slow platelet aggregation Erzhi easy thrombosis; sleep increased vagal tone during coronary artery spasm can be increased to an extent which rendered necrotic myocardial ischemia can occur in patients with frequent angina may also be in the original and in asymptomatic
If it were not itself have had congenital heart disease before myocardial ischemia should be!
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Unknown - Thursday, 28 April 2011
